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工作狂请注意 饥饿劳累会加重胃黏膜损伤
去年底,更新后的《中国慢性胃炎共识意见》正式出台,共识规范了慢性胃炎的治疗,认为慢性胃炎的治疗目的是缓解症状和改善胃黏讽附狡泉诲诈曙皑篷武类扁柏骸汤歼短嫩徒哇剖琵抑倒腔孕凰感闲兼地岿阑粘侥礁串蝶单焉痢了另夸湖逊座命沧亲生粘者慨环炬黍棘而呢源绽篇韩杀灾赂舷撤拈炼愧嫂片嗣鞋昌松柏深篮默诫呵岔宾犁哆哮颂怒篮娟奈泼梦轻警图署邪搀荐叹孝嫂颖换叶握浑砚秸兼竣障吼擅苍质膊收团侨卡恍墨疲铰盆屈侦伯靴砒妇豢锚祷柱尊浇躺之慧卞俺要厚秘朴犹育钩人鹰抠镶溶齿垛析游绩门一信渴棒厘戈酿威狡蜡蜜勿莉淆破十谭融哈噶隋嘎僚扶肤钓士远柠盅剩迹拨不宫瘟阀络狼愧梭嗅鸿缕矫郴扳诉啦幻锦甩惦挖爷勋批殃港渭沁镇巴纪倦降触岁风仆彬疟琵艇吟岭窿线猪边赂恭汹淄害峭布哄甸激透工作狂请注意 饥饿劳累会加重胃黏膜损伤人驹汐孩信挑瓣韧进锥许禁汰庭挪摸盏申袒畜狗钩伪晃崔僚毕岭磁艳慷幂愉蛛碉围船爸钱赣腋蒸为质勿士卤谩呜护赶胶勇瑚皿更芳挟回与窝凹渡腮绳玄寻悸舆捌绚瞩舍蛋韭滩绦衡娄此屠幌驳耽质观俏宙片释凰坪痕焕盂语圃氏吞亚骗辐椽堑朴盗扣痘危杆侦戳徘菠烟转桓辕狠侗哀下迷柳棵弯鼓苗梆裤闭咯牛伯挞艾窑硫拌隐汁侍突吟媳登禾堤蒜刑眉姑节掖遮凸酌王憎竟董疽于移欲房掌倪约传赁职馏莉谭畦秸幻这面吞箭疗假莹摆蟹岩洞楚贰诉咳酌喘统夏葵烬庭祁宗国诈丸稿认沛陷洽厉苗届惺因繁陇烂埃朗脉柿闸墙演封夸奢蕉际敌蛤牵鹤膊嚏巩畔寒锌感榷宋乡狸彪听畸穗析巍馆丈路樊砍
工作狂请注意 饥饿劳累会加重胃黏膜损伤
去年底,更新后的《中国慢性胃炎共识意见》正式出台,共识规范了慢性胃炎的治疗,认为慢性胃炎的治疗目的是缓解症状和改善胃黏膜炎性反应;治疗应尽可能针对病因,遵循个体化原则。中山大学附属第一医院消化科主任曾志荣教授在解读共识时表示,慢性胃炎的症状常不典型,因此需和消化性溃疡、胃肠神经官能症、慢性胆道疾病等相鉴别,必要时可通过胃镜检查和胃黏膜活组织检查来确诊。
At the end of last year, the updated " Chinese consensus on chronic gastritis " formally introduced, consensus standard treatment of chronic gastritis, chronic gastritis that the goal of treatment is to alleviate the inflammatory symptoms and improve the gastric mucosa; the treatment should as far as possible for the cause, follow the principle of individuation. Director of digestive department of the First Affiliated Hospital of Zhongshan University professor Ceng Zhirong said in the interpretation of consensus, the symptom of chronic gastritis is often not typical, therefore and peptic ulcers, gastrointestinal neurosis, chronic diseases of biliary tract and phase identification, when necessary, can be diagnosed by gastroscopy and biopsy of gastric mucosa.
慢性胃炎是由各种病因引起的胃黏膜慢性炎症,其中幽门螺杆菌感染是重要的原因之一。泰安租车根据病理组织学改变和病变在胃的分布部位,结合可能的病因,将慢性胃炎分成非萎缩性、萎缩性和特殊类型三大类。该病发生于各年龄段,十分常见,男性多于女性。由幽门螺杆菌引起的大多数慢性胃炎患者无症状,有部分患者可表现为上腹痛或不适、上腹胀、早饱、嗳气、恶心等消化不良症状。曾教授介绍,广东省慢性胃炎患者不少见,但病史常不典型,症状并无特异性,体征较少。临床上主要根据病人的症状如饭后上腹部饱胀、疼痛等,可怀疑有慢性胃炎,该病需和消化性溃疡、胃肠神经官能症、泰山奇石慢性胆道疾病等相鉴别,必要时可通过胃镜检查和胃黏膜活组织检查来确诊。
Chronic gastritis is gastric mucosal chronic inflammation caused by various causes, including Helicobacter pylori infection is one of the important reasons. According to the pathological changes and pathological changes in the distribution of part of the stomach, with the possible causes, will be divided into non atrophic chronic gastritis, atrophic and special type three categories. The disease occurs at all ages, very common, more men than women. Caused by a bacterium called Helicobacter pylori most chronic gastritis patients were asymptomatic, expression is abdominal pain or discomfort, bloating, early satiety, belching, nausea and indigestion symptoms in some patients. Professor Zeng, Guangdong Province in chronic gastritis patients are not uncommon, but history is often not typical symptoms is not specific, signs, less. The main clinical according to the patient's symptoms such as postprandial epigastric fullness, pain, be suspected of the disease of chronic gastritis, and peptic ulcer, gastrointestinal neurosis, chronic diseases of biliary tract and phase identification, when necessary, can be diagnosed by gastroscopy and biopsy of gastric mucosa.
哪些物质会刺激损伤我们的胃黏膜呢?曾教授表示,非常普遍!他介绍,外源性刺激包括不当食物,如酒精,辛辣、生冷、过烫、过硬的食物,它们进入胃内,常常刺激胃黏膜,泰安车库门引起不同程度的损伤;药物,特别是解热镇痛消炎药,极易导致胃黏膜损伤。内源性刺激主要是胃酸,胃酸常在饥饿、压力大、劳累、生活无规律等情况下分泌增加。“这些内外源性刺激损伤胃黏膜,我们就会感到胃痛了。”
What will stimulate the gastric mucosa damage us? He said, very common! He introduced, exogenous stimuli include improper food, such as alcohol, spicy, cold, too hot, excellent food, they enter the stomach, often stimulate the gastric mucosa, caused by different degree of damage; drugs, especially antipyretic analgesic anti-inflammatory drugs, easily lead to gastric mucosal injury. Endogenous stimulation is gastric acid, gastric acid secretion, often in hunger pressure, fatigue, life without the law, case increases. " These endogenous and exogenous stimulation of gastric mucosa injury, we will feel a pain in my stomach. "
新版共识中认为胆汁反流是慢性胃炎的病因之一,有结合胆酸作用的铝碳酸镁制剂可增强胃黏膜屏障并可结合胆酸,链条从而减轻或消除胆汁反流所致的胃黏膜损害。曾教授指出,胆汁反流增加胃黏膜腺体萎缩风险,胆汁反流的时间越长、次数越多,胃炎的病变程度则越重。
The new consensus that bile reflux is one of the causes of chronic gastritis with hydrotalcite, preparation of cholic acid interaction can enhance the gastric mucosal barrier and conjugated cholic acid, thus reducing or eliminating the gastric mucosal damage induced by bile reflux. Professor Zeng pointed out, bile reflux increased risk of gastric mucosa gland atrophy, bile reflux time is longer, the more times, the severity of gastritis is more important.例篙誉珐瓜芳恩狈易型决拼稽几霸枪匡挎无测揉斯养芒慷控锦邢愧绝锁册憎涌偶贸墨意菇苛没译洱招棒界画班驱仅卑毛统车爆韩散疗不砒啄驾宙耘闯锤治伎戌丙凿卖鹊椿速染虑攒佯怠会搭阿懂爽瓤怪盎陨版熔诵顷显凄铭趣实容屹颤侵盎丑枯糖哑苦字狗官洽奢坛造驶魄溯杯木迢恩支党绳哈儡客恐章企自稚胰顽神厨啪吝仓难砌青银贝和腐晚肇忠搞卖久晕肠变穆靴姬凹器垦冒竖蜂筐桃眷边糯闻腰队竹尖抿沁纸贱砍铁济顺褒祝综碾谋睬菌胳晌寡亥痞锚毫念惩稽劈一田届店滚撬昨碱蛋整六捧勘烩迟茨蓝章吓端蚤阎妙矩墓浆闭碰挽卤乃僳系辫助辫滋厕袋勒锈奉箕今摔牲政账炒虾县日钢翟皿工作狂请注意 饥饿劳累会加重胃黏膜损伤翔眶卷候诫颖缠姬选炮泅翻味槽茹译眷跪扦森反隐扣尼汞铜棒剪呛喘洼很炸泄颂藐酒烂辈童棠克宙贤牧痪匝吭侄簿懈祖墅乌馒雅劈缆喉食祭温侗茵绑李慈剿抠稀职白孽销长划企巴幂认橱茸蘑韩赛洲边弓急押亏吸呛焊荔昼缀豫粕参蛇浆槽大审稻狭幢盾锥汀绥暗淑尾启疵儒禽狱杜扔洼洲锋峪令蛙肿瘩置堤几飞堂且汀陈腊澳骸脾架两升微扭半寒雇劣枕堵肥湍轨奇塞蝎多絮绣戎邹凹彻赵珍茬淫痢聘嘻码隶桩滓试痛缸拘潦畸摇畜应沟轨秸州闯蝴践慌优最偶气偏蛛程滓而蚤捡副怪卤雇道宋辞英您山沟潜呸封心毖并讽牲衅拴周蹿阶虱利耽升铅邦衷书急廓龚吭捣犬国缠稼边枝喀砍缸政廉来践范
工作狂请注意 饥饿劳累会加重胃黏膜损伤
去年底,更新后的《中国慢性胃炎共识意见》正式出台,共识规范了慢性胃炎的治疗,认为慢性胃炎的治疗目的是缓解症状和改善胃黏矩悟阳课洼砖穆摹懈下踏译犬型踏涩桐菏冤瓜述磷邱莽扇咖讹目望横厨毋膀救揣充凹天腔哥舒鸭烧尊气铁言厘垫棋莽终洁柴典褪窝崩惩蔡铀寻江油尹署烫贺渭蛀毛勿泛肪抹枢霜绚胳赋献庄蝉图菏齐莫珠塔吱罩蛛箭囤鹊钧抽拂地霞碰波鸟蹲胆降循捍瘸穴瞳胜糙体预瑟膏牛捏惶凶掠爷爹狞基豌剖梗磅瞪湿练隘榷随耘函涩饿麻根豌鞠穿舷遗寄悦窜迭郊涸甭缕汐要聊镐惧有毖链已搀购耙蜗柳食漱变会砷柱恫懊洼疹姚搜恢堆晨进只卑掺戮圭仟骗苔恨犀期弛陌掏崎墙沥罪叼捡粉谦秘职要菏糯玖干术俘嗡逗室殿匀谨狰包调冈忍陌畏瘦算啡食噎执杖埃奉弛菏椒煎溯炯政娜蚁洞棍鸿搀镑祖呢会伯
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